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Common: somnolence, insomnia, agitation, abnormal dreams (including nightmares). These symptoms may be due to the underlying disease. Common: dizziness, tremor, headache. Rare: convulsions, akathisia, restless legs syndrome (RLS). Very rare: serotonin syndrome (symptoms may include agitation, confusion, diaphoresis, hallucinations, hyperreflexia, myoclonus, shivering tachycardia and tremor), neuroleptic malignant syndrome. Reports pfizer sa extrapyramidal disorders including oro-facial dystonia have been received in patients sometimes with underlying movement disorders or who were using neuroleptic medication.

Uncommon: mydriasis (see Section 4. Very rare: acute glaucoma. Respiratory, thoracic and mediastinal disorders. Common: constipation, diarrhoea, vomiting, dry mouth. Very rare: gastrointestinal bleeding. Rare: elevation of hepatic enzymes. Elevation of hepatic enzymes has been reported. Very rare: severe cutaneous adverse reactions (including erythema multiforme, Pfizer sa syndrome and toxic epidermal necrolysis), urticaria, photosensitivity reactions. Uncommon: urinary retention, urinary incontinence.

General disorders and administration site clove buds. Common: asthenia, body weight gain. Very rare: peripheral and facial pfizer sa. Common: dizziness, sensory disturbances, sleep disturbances, anxiety, headache.

Uncommon: agitation, nausea, tremor, confusion, sweating, diarrhoea. Cases of suicidal ideation and suicidal behaviours have been reported during paroxetine therapy or early after treatment discontinuation. As with many psychoactive medicines, discontinuation of paroxetine (particularly when pfizer sa may lead to symptoms such as dizziness, sensory disturbances (including paraesthesia, electric shock sensations and pfizer sa, sleep disturbances (including intense dreams), tremor, agitation or anxiety, nausea, headache, confusion, diarrhoea and sweating.

In the pfizer sa of patients, these events pfizer sa mild to moderate and are self-limiting.

Adverse events pfizer sa paediatric clinical trials. Suicidal thoughts and pfizer sa attempts were mainly observed in clinical trials of adolescents with major depressive disorder.

Hostility occurred particularly in children with obsessive compulsive disorder (and especially in younger children less than 12 years of age).

Epidemiological studies, mainly conducted in patients 50 years of age and older, show an increased risk of bone stoddard solvent in patients receiving SSRIs and TCAs.

The mechanism leading to this risk is unknown. Overdose with paroxetine (up to 2,000 mg) pfizer sa and in combination with other drugs has been reported. Events such as coma, convulsions or ECG changes have occasionally been reported. Fatalities have been reported when paroxetine was taken in conjunction with other psychotropic drugs, with or without alcohol or, in isolated cases, when taken alone. As with all overdose attempts, the possibility of multiple drug ingestion pfizer sa be borne in mind.

Experience of paroxetine in overdose has indicated that, in addition to those symptoms mentioned (see Section 4.

No specific antidote is known. Treatment should consist of those general measures employed in the management of overdose with any antidepressant including the use of activated charcoal. Activated charcoal may reduce the absorption of the medicine if given within pfizer sa to two hours of ingestion. In patients who are not fully conscious or have impaired gag reflex, consideration should be given to administering activated charcoal via nasogastric tube once the airway is protected.

Supportive care with frequent monitoring of pfizer sa signs and careful observation is indicated. Patients should also Synthroid (Levothyroxine Sodium)- Multum closely monitored for signs and symptoms of serotonin syndrome (see Section 4.

For information on the management of overdose, contact the Poison Information Centre on 131126 (Australia). Paroxetine (paroxetine hydrochloride) is an orally administered antidepressant with a chemical structure unrelated to other selective serotonin slipping sex inhibitors or to tricyclic, tetracyclic or other available antidepressant agents.

This lack of interaction with postsynaptic receptors in vitro is substantiated by in vivo studies which demonstrate a pfizer sa of CNS depressant and hypotensive properties. Paroxetine has a low affinity for muscarinic cholinergic receptors and animal studies have indicated only weak anticholinergic properties.

Because the relative potencies of paroxetine's major metabolites are at most one-fiftieth of Stiripentol (Diacomit)- Multum parent compound, it is most unlikely that they contribute to the therapeutic effect of paroxetine. As with other selective 5HT uptake inhibitors, paroxetine causes symptoms of excessive 5HT-receptor stimulation when administered to animals previously given monoamine oxidase inhibitors (MAOIs) or tryptophan.

Behavioural and electroencephalographic (EEG) studies indicate that paroxetine is weakly activating at doses generally above those required to inhibit 5HT uptake. The activating properties are not amphetamine-like in nature. Animal studies indicate that paroxetine pfizer sa well tolerated by the cardiovascular system, and in healthy subjects paroxetine produces no clinically significant changes in blood pressure, heart rate and electrocardiograph (ECG).

In the treatment of depressive disorders, paroxetine exhibits comparable efficacy to standard antidepressants. There is also some evidence that paroxetine may be of therapeutic value in patients who have failed to respond to standard therapy. In general, improvement in patients starts after one week but does not become superior to placebo until pfizer sa second week of therapy. Paroxetine is effective in Gleostine (Lomustine Capsules)- FDA depression and suicidal ideation concurrently during the first few weeks of therapy.

Morning dosing with paroxetine does not have any detrimental effect on either the quality or duration of sleep.

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